Mouse Anti-MME (Dako Omnis) Recombinant Antibody (CB058) (CBMAB-AL058LY)
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Basic Information
Formulations & Storage [For reference only, actual COA shall prevail!]
Target
Biologically important in the destruction of opioid peptides such as Met- and Leu-enkephalins by cleavage of a Gly-Phe bond (PubMed:17101991).
Able to cleave angiotensin-1, angiotensin-2 and angiotensin 1-9 (PubMed:15283675).
Involved in the degradation of atrial natriuretic factor (ANF) and brain natriuretic factor (BNP(1-32)) (PubMed:2531377, PubMed:2972276, PubMed:16254193).
Displays UV-inducible elastase activity toward skin preelastic and elastic fibers (PubMed:20876573).
Amyloid-beta clearance Source: ARUK-UCL
Amyloid-beta clearance by cellular catabolic process Source: ARUK-UCL
Amyloid-beta metabolic process Source: UniProtKB
Cellular response to cytokine stimulus Source: UniProtKB
Cellular response to UV-A Source: UniProtKB
Cellular response to UV-B Source: UniProtKB
Creatinine metabolic process Source: UniProtKB
Kidney development Source: UniProtKB
Learning or memory Source: ARUK-UCL
Lung development Source: Ensembl
Neuropeptide processing Source: ARUK-UCL
Peptide metabolic process Source: UniProtKB
Placenta development Source: Ensembl
Positive regulation of long-term synaptic potentiation Source: Ensembl
Positive regulation of neurogenesis Source: ARUK-UCL
Protein processing Source: GO_Central
Proteolysis Source: UniProtKB
Replicative senescence Source: UniProtKB
Sensory perception of pain Source: UniProtKB
An axonal form of Charcot-Marie-Tooth disease, a disorder of the peripheral nervous system, characterized by progressive weakness and atrophy, initially of the peroneal muscles and later of the distal muscles of the arms. Charcot-Marie-Tooth disease is classified in two main groups on the basis of electrophysiologic properties and histopathology: primary peripheral demyelinating neuropathies (designated CMT1 when they are dominantly inherited) and primary peripheral axonal neuropathies (CMT2). Neuropathies of the CMT2 group are characterized by signs of axonal degeneration in the absence of obvious myelin alterations, normal or slightly reduced nerve conduction velocities, and progressive distal muscle weakness and atrophy.
Spinocerebellar ataxia 43 (SCA43):
A form of spinocerebellar ataxia, a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA43 is a slowly progressive, autosomal dominant form.
Helical: 29-51
Extracellular: 52-750
Glycosylation at Asn-628 is necessary both for surface expression and neutral endopeptidase activity.
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Please try the standard protocols which include: protocols, troubleshooting and guide.
Enzyme-linked Immunosorbent Assay (ELISA)
Flow Cytometry
Immunofluorescence (IF)
Immunohistochemistry (IHC)
Immunoprecipitation (IP)
Western Blot (WB)
Enzyme-Linked Immunospot (ELISpot)
Proteogenomics
Other Protocols
Custom Antibody Labeling
We also offer labeled antibodies developed using our catalog antibody products and nonfluorescent conjugates (HRP, AP, Biotin, etc.) or fluorescent conjugates (Alexa Fluor, FITC, TRITC, Rhodamine, Texas Red, R-PE, APC, Qdot Probes, Pacific Dyes, etc.).
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