Mouse Anti-HBS1L (AA 1-242) Recombinant Antibody (CBFYH-0759) (CBMAB-H1664-FY)

Chumchuen, S., Sripichai, O., Jearawiriyapaisarn, N., Fucharoen, S., & Peerapittayamongkol, C. (2023). Induction of fetal hemoglobin: Lentiviral shRNA knockdown of HBS1L in β0-thalassemia/HbE erythroid cells. Plos one, 18(3), e0281059.

Sales, R. R., Nogueira, B. L., Belisário, A. R., Faria, G., Mendes, F., Viana, M. B., & Luizon, M. R. (2022). Fetal hemoglobin-boosting haplotypes of BCL11A gene and HBS1L-MYB intergenic region in the prediction of clinical and hematological outcomes in a cohort of children with sickle cell anemia. Journal of Human Genetics, 67(12), 701-709.

Mohammad, S. N. N. A. I., Iberahim, S., Wan Ab Rahman, W. S., Hassan, M. N., Edinur, H. A., Azlan, M., & Zulkafli, Z. (2022). Single Nucleotide Polymorphisms in XMN1-HBG2, HBS1L-MYB, and BCL11A and Their Relation to High Fetal Hemoglobin Levels That Alleviate Anemia. Diagnostics, 12(6), 1374.

Bashir, S., Mahmood, S., Mohsin, S., Tabassum, I., Ghafoor, M., & Sajjad, O. (2021). Modulatory effect of single nucleotide polymorphism in Xmn1, BCL11A and HBS1L-MYB loci on foetal haemoglobin levels in β-thalassemia major and Intermedia patients. J. Pak. Med. Assoc, 71, 1394-1398.

Akbulut-Jeradi, N., Fernandez, M. J., Al Khaldi, R., Sukumaran, J., & Adekile, A. (2021). Unique polymorphisms at BCL11A, HBS1L-MYB and HBB loci associated with HbF in Kuwaiti patients with sickle cell disease. Journal of personalized medicine, 11(6), 567.

Sales, R. R., Belisário, A. R., Faria, G., Mendes, F., Luizon, M. R., & Viana, M. B. (2020). Functional polymorphisms of BCL11A and HBS1L-MYB genes affect both fetal hemoglobin level and clinical outcomes in a cohort of children with sickle cell anemia. Annals of Hematology, 99(7), 1453-1463.

Yang, K., Wu, Y., Ma, Y., Xiao, J., Zhou, Y., & Yin, X. (2020). The association of HBG2, BCL11A, and HBS1L-MYB polymorphisms to thalidomide response in Chinese β-thalassemia patients. Blood Cells, Molecules, and Diseases, 84, 102442.

O’Connell, A. E., Gerashchenko, M. V., O’Donohue, M. F., Rosen, S. M., Huntzinger, E., Gleeson, D., ... & Agrawal, P. B. (2019). Mammalian Hbs1L deficiency causes congenital anomalies and developmental delay associated with Pelota depletion and 80S monosome accumulation. PLoS genetics, 15(2), e1007917.

Morrison, T. A., Wilcox, I., Luo, H. Y., Farrell, J. J., Kurita, R., Nakamura, Y., ... & Chui, D. H. (2018). A long noncoding RNA from the HBS1L-MYB intergenic region on chr6q23 regulates human fetal hemoglobin expression. Blood Cells, Molecules, and Diseases, 69, 1-9.

Trifa, A. P., Bănescu, C., Bojan, A. S., Voina, C. M., Popa, Ș., Vișan, S., ... & Popp, R. A. (2018). MECOM, HBS1L‐MYB, THRB‐RARB, JAK2, and TERT polymorphisms defining the genetic predisposition to myeloproliferative neoplasms: a study on 939 patients. American Journal of Hematology, 93(1), 100-106.

Please try the standard protocols which include: protocols, troubleshooting and guide.

Enzyme-linked Immunosorbent Assay (ELISA)
Flow Cytometry
Immunofluorescence (IF)
Immunohistochemistry (IHC)
Immunoprecipitation (IP)
Western Blot (WB)
Enzyme-Linked Immunospot (ELISpot)
Proteogenomics
Other Protocols