Human Recombinant VAPB protein, His Tag (V2LY-0526-LY7582)
Compare
Online Inquiry
Basic Information
Formulations & Storage [For reference only, actual COA shall prevail!]
Target
Biological Process COPII-coated vesicle budding Source:UniProtKB1 Publication
Biological Process endoplasmic reticulum membrane organization Source:GO_Central1 Publication
Biological Process endoplasmic reticulum organization Source:UniProtKB1 Publication
Biological Process endoplasmic reticulum to Golgi vesicle-mediated transport Source:UniProtKB1 Publication
Biological Process endoplasmic reticulum unfolded protein response Source:UniProtKB1 Publication
Biological Process endoplasmic reticulum-plasma membrane tethering Source:GO_Central1 Publication
Biological Process IRE1-mediated unfolded protein response Source:UniProtKB1 Publication
Biological Process modulation by host of viral RNA genome replication Source:AgBase
Biological Process negative regulation by host of viral genome replication Source:AgBase
Biological Process negative regulation by virus of viral protein levels in host cell Source:AgBase
Biological Process positive regulation by host of viral genome replication Source:AgBase
Biological Process positive regulation of viral genome replication Source:UniProtKB1 Publication
Biological Process suppression of viral release by host Source:AgBase
Biological Process viral release from host cell Source:AgBase
Present in mitochondria-associated membranes that are endoplasmic reticulum membrane regions closely apposed to the outer mitochondrial membrane.
A neurodegenerative disorder affecting upper motor neurons in the brain and lower motor neurons in the brain stem and spinal cord, resulting in fatal paralysis. Sensory abnormalities are absent. The pathologic hallmarks of the disease include pallor of the corticospinal tract due to loss of motor neurons, presence of ubiquitin-positive inclusions within surviving motor neurons, and deposition of pathologic aggregates. The etiology of amyotrophic lateral sclerosis is likely to be multifactorial, involving both genetic and environmental factors. The disease is inherited in 5-10% of the cases.
Spinal muscular atrophy, proximal, adult, autosomal dominant (SMAPAD):
A form of spinal muscular atrophy, a neuromuscular disorder characterized by degeneration of the anterior horn cells of the spinal cord, leading to symmetrical muscle weakness and atrophy. SMAPAD is characterized by proximal muscle weakness that begins in the lower limbs and then progresses to upper limbs, onset in late adulthood (after third decade) and a benign course. Most of the patients remain ambulatory 10 to 40 years after clinical onset.
Helical: 223-243
Submit a review and get a Coupon or an Amazon gift card. 20% off Coupon
$30 eGift Card
Submit a review
Loading...
Please try the standard protocols which include: protocols, troubleshooting and guide.
Enzyme-linked Immunosorbent Assay (ELISA)
Flow Cytometry
Immunofluorescence (IF)
Immunohistochemistry (IHC)
Immunoprecipitation (IP)
Western Blot (WB)
Enzyme Linked Immunospot (ELISpot)
Proteogenomic
Other Protocols
Custom Antibody Labeling
We also offer labeled antibodies developed using our catalog antibody products and nonfluorescent conjugates (HRP, AP, Biotin, etc.) or fluorescent conjugates (Alexa Fluor, FITC, TRITC, Rhodamine, Texas Red, R-PE, APC, Qdot Probes, Pacific Dyes, etc.).
Online InquiryContact us
