Mouse Anti-FBXO28 Recombinant Antibody (3E5) (CBMAB-A2885-LY)

Phillips, E., Balss, J., Bethke, F., Pusch, S., Christen, S., Hielscher, T., ... & Goidts, V. (2022). PFKFB4 interacts with FBXO28 to promote HIF-1α signaling in glioblastoma. Oncogenesis, 11(1), 1-12.

Schneider, A. L., Myers, C. T., Muir, A. M., Calvert, S., Basinger, A., Perry, M. S., ... & Scheffer, I. E. (2021). FBXO28 causes developmental and epileptic encephalopathy with profound intellectual disability. Epilepsia, 62(1), e13-e21.

Cai, L., Liu, L., Li, L., & Jia, L. (2020). SCFFBXO28-mediated self-ubiquitination of FBXO28 promotes its degradation. Cellular Signalling, 65, 109440.

Yu, T., Wang, L., Zhao, C., Qian, B., Yao, C., He, F., ... & Qiu, W. (2019). Sublytic C5b‐9 induces proliferation of glomerular mesangial cells via ERK5/MZF1/RGC‐32 axis activated by FBXO28‐TRAF6 complex. Journal of cellular and molecular medicine, 23(8), 5654-5671.

Yanagishita, T., Yamamoto-Shimojima, K., Nakano, S., Sasaki, T., Shigematsu, H., Imai, K., & Yamamoto, T. (2019). Phenotypic features of 1q41q42 microdeletion including WDR26 and FBXO28 are clinically recognizable: the first case from Japan. Brain and Development, 41(5), 452-455.

Balak, C., Belnap, N., Ramsey, K., Joss, S., Devriendt, K., Naymik, M., ... & Narayanan, V. (2018). A novel FBXO28 frameshift mutation in a child with developmental delay, dysmorphic features, and intractable epilepsy: A second gene that may contribute to the 1q41‐q42 deletion phenotype. American Journal of Medical Genetics Part A, 176(7), 1549-1558.

Please try the standard protocols which include: protocols, troubleshooting and guide.

Enzyme-linked Immunosorbent Assay (ELISA)
Flow Cytometry
Immunofluorescence (IF)
Immunohistochemistry (IHC)
Immunoprecipitation (IP)
Western Blot (WB)
Enzyme Linked Immunospot (ELISpot)
Proteogenomic
Other Protocols